The use of Botox in urology is now approved by the FDA and many studies have found it’s useful potential in relieving a variety of lower urinary tract dysfunctions including multiple sclerosis, stroke, overactive bladder, interstitial cystitis, and spinal cord injury when conventional treatments have failed.
Botox works by paralyzing the nerves that stimulate the bladder to contract. By preventing release of the chemicals responsible for stimulating bladder contraction, the bladder muscle weakens thereby reducing the spasticity. Studies have shown that following injection of Botox into the bladder, there was a decrease in urinary urgency, frequency, and leakage episodes of approximately 70% to 80%.
Possible side effects of using Botox include urinary tract infection, bleeding, and difficulty emptying your bladder. Your physician may recommend that you learn to self-catheterize in the event that you are not able to urinate after your Botox injection, if you do not already have an indwelling catheter in place. As of yet, there have been no negative systemic affects reported when using Botox in the bladder.